Inflammation and free radicals: • Inflammation is part of the body’s natural defense against irritation‚ toxic foods and chemicals‚ infections and other foreign invaders. However‚ there are times when the appropriate immune response to control infections‚ allergies and toxins is disrupted. The immune system can then shift into a chronic state of inflammation‚ which is now recognized as a major factor in most chronic health problems. Free radicals are produced during normal metabolism and cell function
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Essential of Pathophysiology I LP1 Essay Inflammation What is inflammation? This is term that is used to describe a reaction of localized tissues that is irritated‚ or can be cause because of an injury‚ infection‚ redness and swelling. There are several types of disease that cause severe inflammation‚ one disease that is very bad is Encephalitis this is an inflammation of the brain. This particular condition can happen to anyone at any age‚ all over the
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Acute Inflammation The survival of all organisms requires that they eliminate foreign invaders‚ such as infectious pathogens‚ and damaged tissues. These functions are mediated by a complex host response called inflammation. Definition of inflammation Inflammation is fundamentally a protective response‚ the ultimate goal of which is to rid the organism of both the initial cause of cell injury (e.g.‚ microbes‚ toxins) and the consequences of such injury (e.g.‚ necrotic cells and tissues)
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Inflammation is a protective response by the body towards cell injury‚ irritation‚ infection and can accumulate fluids with the following signs and symptoms: redness‚ warmth‚ swelling and pain. Inflammation is therefore the body’s way of attempting to remove the primary cause of inflammation and any damage that may have occurred as a result (healing and repair) (Drake et al.‚ 2007). Today numerous drugs are used to treat conditions of inflammation such as narcotics (opioids)‚ and non narcotics (salicylates)
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Wound care (Pressure Ulcer) Descriptions During community placement‚ my mentor and I visited M (patient)‚ a 75years old lady‚ who was presented with a Pressure Ulcer‚ on the heel of her right leg. On arrival‚ my mentor asked me to manage M’s wound. However‚ I have observed and participate in carrying out this skill (wound care) with my mentor on several occasions. I explained the procedure to M and gained her consent to carry out the procedure. The preparation and application of aseptic technique
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4 Stages of Healing How Do Wounds Heal? Research work on acute wounds in an animal model shows that wounds heal in four phases. It is believed that chronic wounds must also go through the same basic phases. Some authors combine the first two phases. The phases of wound healing are: * Hemostasis * Inflammation * Proliferation or Granulation * Remodeling or Maturation Hemostasis: Once the source of damage to a house has been removed and before work can start‚ utility workers
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a Break and the Power of Healing What is a Wound? A wound may be described in many ways; by its aetiology‚ anatomical location‚ by whether it is acute or chronic1‚ by the method of closure‚ by its presenting symptoms or indeed by the appearance of the predominant tissue types in the wound bed. All definitions serve a critical purpose in the assessment and appropriate management of the wound through to symptom resolution or‚ if viable‚ healing. A wound by true definition is a breakdown in the protective
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underlying factors that can slow wound healing. Such lab work includes but is not limited to a complete blood count (CBC)‚ and Hemoglobin A1C (HBA1C) which looks at one’s level of glucose over the past three months. In addition‚ an albumin and pre-albumin level should be obtained to look at the amount of protein in your body. If levels such as glucose or white blood cells are high healing will be delayed. On the other hand‚ if one’s hemoglobin and hematocrit are low‚ the wound may not receive the oxygen
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Inflammation is defined as the presence of edema fluid and the invasion of tissue by leukocytes. Inflammation is characterized by heat‚ redness‚ pain‚ and swelling. These characteristics are the result of the release of various chemical mediators during an immune response. Cytokines and other inflammatory mediators act on local blood vessels causing dilation of blood vessels and an increase in vascular permeability. Blood flow is increased and there is an increase in the leakage of fluid and proteins
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syndromes and stroke. Recent advances in cellular science have established a fundamental role for inflammation in mediating all stages of this disease‚ from initiation through progression‚ and ultimately‚ the thrombotic complications of atherosclerosis. These findings are providing important links between predisposing risk factors‚ and the mechanism of atherogenesis. The chronic inflammatory process involving the arterial endothelium‚ that ultimately results in the complications of atherosclerosis
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