Since his birth on June 5th‚ 1932‚ Christy Brown had to dig himself out of a hole‚ using only his left foot. Faced with Cerebral Palsy as a baby‚ he was thought to be mentally challenged‚ and no doctor could do anything to help. His disability was the backbone of the many problems in his life‚ such as his alcoholic father‚ a poor lifestyle‚ as well as some social issues. Growing up‚ his only hope of someday being recognised as a real person rested within his mother. Although‚ after drawing the letter
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Running head: ADDISON ’S DISEASE 1 Addison ’s Disease ADDISON ’S DISEASE 2 Abstract Addison ’s disease is a disorder that occurs when a person ’s adrenal glands produce insufficient amounts of certain hormones. When Addison ’s diseased was first discovered the endocrine system was not yet completely
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responses. The endocrine system consists of the HPA axis that is more conditioned for long-term stressors. The physiological response process begins when the hypothalamus triggers a factor that inhibits the pituitary gland to secrete ACTH. The adrenal cortex is then aroused by ACTH to secrete cortisol‚ which increases blood sugar levels and metabolism. When metabolic activity is increased in the hippocampus‚ neurons become more sensitive to toxins or over-stimulation. This is a slow process‚ but in
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Student’s Name: Marylene Mosburg Date: 11/31/2014 BIO169 Case_10: Nicholas’s Mysterious Suntan This case study presents a clinical situation related to the endocrine system. The questions at the each segment of the case will test your understanding of the endocrine system. Nicholas T.‚ age 32‚ is a busy systems analyst for a computer manufacturer. Working his way up the corporate ladder‚ he travels extensively‚ putting in many hours without much sleep. Nicholas has a tendency to be overweight
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discovered that the RP (which they named BP1) occurred before ‘W’ in two thirds of participants and the LRP always occurred after ‘W’. Klemm (2016) also argued that no one really knows where the conscious self lies within the brain‚ and that the motor cortex only began to increase in activity before the self-reported intent to
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maintains lactation by the mammary glands after childbirth. It may stimulate testerone production in males. Influences the growth and activity of the thyroid gland Stimulates the secretion of glucocorticoids and other hormones of the adrenal cortex In females FSH stimulates maturation of the ovarian follicles and production of estrogen. LH triggers ovulation and stimulates the production of estrogen and progesterone In males FSH stimulates sperm production and LH stimulates production
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increasing which then results in cognition becoming distorted. With distorted cognition‚ people begin to simply see things in black and white. This black and white thinking causes for the arguments to further themselves. b. How does the prefrontal cortex shift in adolescence?
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deformities that I’ve never even heard of. All of it has to do with the brain development mainly. If something were to go wrong with you and it could be just a small little incident that occurs with your brain then it goes all down hill from there. Cerebral Palsy appears in infancy to early childhood and permanently affects the body movement and muscles‚ but it doesn’t worse over time. Some people think that this deformity is caused by problems of the muscle or nerves‚ but it’s not. It is actually caused
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of 1st Guest Speaker 1:25 – 2:10 Lecture on Cerebral Palsy : Medical Perspective By Dr. Annah Rebecca V. Doroja‚ DPPS‚ FPSDBP 2:10 – 2:20 Open Forum 2:20 – 2:30 Synthesis of the Discussion 2:30 – 2:35 Awarding of Plaque and Token of Appreciation By Mrs. Melanie Joy Bustamante‚ RN and Ms. Julie Vira Chua 2:35 – 2:45 Intermission No. 1 2:45 – 2:50 Introduction of 2nd Guest Speaker 2:50 – 3:35 Lecture on Therapies Available for Cerebral Palsy By Mr. John Teodoro‚ PTRP‚ RN 3:35 – 3:45
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INTRODUCTION Congenital Adrenal Hyperplasia (CAH) refers to a complex series of rare but well-studied enzymatic errors of metabolism with deficient levels of different enzymes involved in the synthesis of cortisol (hydrocortisone). CAH comprises a group of disorders resulting from defective synthesis of adrenal corticosteroids. Lack of glucocorticoids‚ especially cortisol‚ causes various kinds of metabolic problems. The response to low levels of cortisol is increased production of corticotropin
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