Daphnia Caffeine Experiment
A-LEVEL
BIOLOGY LAB REPORT
NAME: NURUL AYUNI BT SABRI
GROUP: 11SC4
TITLE: THE EFFECT OF CAFFEINE ON HEART RATE
LECTURER : MDM ZAKIAH BINTI ZAKARIA
TITLE
The effect of caffeine on heart rate OBJECTIVE
To determine the effect of caffeine on heart rate of daphnia
INTRODUCTION
Caffeine, medically known as trimethylzanthine. Its chemical formula is C8H10N4O2. When in pure form, caffeine is a white crystalline powder that taste very bitter. 1The most common way of acquiring pure caffeine is the process of decaffeinating coffee and tea. Caffeine is found in varying quantities in the seeds, leaves, and fruit of some plants, where it acts as a natural pesticide that paralyzes and kills certain insects …show more content…
In contrast, heart rate is increased by the sympathetic nervous system (neurotransmitter: noradrenaline) and the hormone adrenaline circulating in the blood via activation of cell surface receptors in the sinoatrial node - pacemaker) (called beta-1 adrenoceptors). This occurs during exercise or fear. The effect is to increase the rate of ejection of blood by the heart. This means that there will be more blood flow to skeletal muscle (in which exercise causes dilatation of blood vessels), so the skeletal muscle cells are supplied with more oxygen and respiratory substrates used to generate energy in respiration where it is needed. A drug that increases heart rate is called a positive chronotrope, and this is demonstrated in this experiment when adrenaline is used to increase heart rate in Daphnia.
One of the ways adrenaline increases heart rate is through the action of what is known as a 'second messenger ' or 'transduction component ', in this case it is a chemical made in the cell known as cyclic adenosine monophosphate (cAMP). Transduction is the process that follows the action of a drug, hormone or neurotransmitter at a receptor. Thus, when adrenaline activates the beta-1 adrenoceptor in the sinoatrial node, this leads to an increase in cAMP in the sinoatrial node and the result is an increase in heart
BIOLOGY LAB REPORT
NAME: NURUL AYUNI BT SABRI
GROUP: 11SC4
TITLE: THE EFFECT OF CAFFEINE ON HEART RATE
LECTURER : MDM ZAKIAH BINTI ZAKARIA
TITLE
The effect of caffeine on heart rate OBJECTIVE
To determine the effect of caffeine on heart rate of daphnia
INTRODUCTION
Caffeine, medically known as trimethylzanthine. Its chemical formula is C8H10N4O2. When in pure form, caffeine is a white crystalline powder that taste very bitter. 1The most common way of acquiring pure caffeine is the process of decaffeinating coffee and tea. Caffeine is found in varying quantities in the seeds, leaves, and fruit of some plants, where it acts as a natural pesticide that paralyzes and kills certain insects …show more content…
In contrast, heart rate is increased by the sympathetic nervous system (neurotransmitter: noradrenaline) and the hormone adrenaline circulating in the blood via activation of cell surface receptors in the sinoatrial node - pacemaker) (called beta-1 adrenoceptors). This occurs during exercise or fear. The effect is to increase the rate of ejection of blood by the heart. This means that there will be more blood flow to skeletal muscle (in which exercise causes dilatation of blood vessels), so the skeletal muscle cells are supplied with more oxygen and respiratory substrates used to generate energy in respiration where it is needed. A drug that increases heart rate is called a positive chronotrope, and this is demonstrated in this experiment when adrenaline is used to increase heart rate in Daphnia.
One of the ways adrenaline increases heart rate is through the action of what is known as a 'second messenger ' or 'transduction component ', in this case it is a chemical made in the cell known as cyclic adenosine monophosphate (cAMP). Transduction is the process that follows the action of a drug, hormone or neurotransmitter at a receptor. Thus, when adrenaline activates the beta-1 adrenoceptor in the sinoatrial node, this leads to an increase in cAMP in the sinoatrial node and the result is an increase in heart